The role of TLR4 in brainischemia has not been examined yet.
2
Enhanced glucose uptake protects cells during energy depletion including brainischemia.
3
Summary: Reversible brainischemia is a harbinger for subsequent ischemic stroke.
4
Four-vessel occlusion model of transient global brainischemia was used.
5
PGA1 treatment also significantly ameliorated motor dysfunction after brainischemia.
1
To evaluate their prognostic value, five different non-invasive techniques were used on 34 full term infants with hypoxic- ischaemicencephalopathy (HIE) within six hours of delivery.
1
Ranibizumab was not associated with any cases of cerebrovascular hemorrhage and cerebrovascularischemia.
2
The presence of TCD-detected microemboli could be a risk factor for cerebrovascularischemia.
3
Conclusions: These data suggest that TCD-detected microemboli are associated with an increased prevalence of prior cerebrovascularischemia.
1
The outcomes of hypoxic- ischemicencephalopathy vary between death and intact survival.
2
We conclude that diffusion tensor imaging can quantify white matter injury in neonates with hypoxic- ischemicencephalopathy.
3
His death certificate lists the cause of death as hypoxic ischemicencephalopathy caused by cardiopulmonary arrest.
4
Outcome measures consisted of previously reported indices that were modified including the addition of hypoxic ischemicencephalopathy.
5
This makes it increasingly important to assess, soon after birth, the prognosis of children with hypoxic- ischemicencephalopathy.
Uso de cerebral ischemia en inglés
1
However, the mechanisms underlying its neuroprotective effects following cerebralischemia remain unclear.
2
These data suggest that general anesthesia increased the tolerance to cerebralischemia.
3
Rehabilitation therapy through physical exercise also provides therapeutic efficacy for cerebralischemia.
4
These findings suggest that baicalin has a neuroprotective effect on cerebralischemia.
5
However, selective neuronal death in transient focal cerebralischemia needs more investigation.
6
Temporary bypass for maintaining cerebral perfusion was useful in preventing cerebralischemia.
7
The complement anaphylatoxin C3a contributes to injury after cerebralischemia in mice.
8
Evidence suggests that apoptosis contributes significantly to cell death after cerebralischemia.
9
Increased tolerance to cerebralischemia produced by general anesthesia during temporary carotid occlusion.
10
These results suggest that DMSO is neuroprotective in focal cerebralischemia.
11
Global cerebralischemia was performed via 10 min of four-vessel occlusion.
12
Focal cerebralischemia elicits a homogeneous expression of several IEGs, prominently in cortex.
13
Outcome from cerebralischemia is sexually dimorphic in many experimental models.
14
The patient has experienced no further episode of cerebralischemia or intracranial bleeding.
15
Programmed cell death is an important contributor to neuronal loss following cerebralischemia.
16
TFA has shown neuroprotective effect against cerebralischemia injury in rats and rabbits.